Understanding Second-Degree Burns and Healing
BIOL2001C - Anatomy and Physiology I
University of Cincinnati
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
When Matt goes to Miami for spring break, he experiences severe sunburn that causes hislegs
and arms to become red, swollen, and painful. The next day, he develops blisters in the afflicted
areas that cover the front and back of his trunk, arms, and legs. In this scenario, Matt has
damaged his skin, an organ part of the integumentary system. According to John Hopkin’s
medicine, Matt’s symptomology of blisters associated with swelling, redness, and pain
classifies him as having a second-degree burn. A second-degree burn involves the epidermis
and dermis layers of the skin (Johns Hopkins Medicine, 2023). The tissues of the skin that have
been afflicted include the stratified squamous epithelial layer of the epidermis (Cleveland
Clinic, 2021). The burn also extends into the dermis layer, which damages the dense irregular
connective tissue that makes up the dermis (Cleveland Clinic, 2022). Matt experienced a
second-degree burn, which is indicated by the blistering that was noted on his body. These
blisters form due to an inflammatory response to increase capillary permeability, also known
as the neovascularization phase. At this stage, the blisters stimulate healing since the fluid
associated with blisters has high growth factors and facilitates fibroblast growth. This burn
wound healing can be divided into three phases: the inflammation phase, the proliferative
phase, and matrix remodeling. Matt is currently in the inflammation phase due to the presence
of blisters. Soon, he will enter the proliferative phase in which reepithelization occurs (Pan,
2013).
The skin of the integumentary system is responsible for various bodily functions such as
protection, body temperature regulation, detection of stimuli, and synthesis of vitamin D (Dao;
Kim, 2023). Due to Matt’s second-degree burn extending past the epidermis into the dermis
layerof the skin, some of these bodily functions have become dysregulated. This burn
ultimately weakens the immune system as the skin cannot act as a protection barrier against
pathogens, which increases the risk for body infections. Furthermore, thermal regulation is
inhibited, and thedamaged part of the body loses its ability to sweat, which makes it harder for
Matt to withstand hotter climates. Lastly, the sensation of stimuli to this area is heightened due
to the damaged nerve endings, so any touch or movement may be painful; therefore, certain
activities and mobility may be restrictive (Schaefer; Tannan, 2023). After a couple of days,
Matt’s skin starts to peel, and the burns begin to heal. However, Matt experiences increased
susceptibility to injury from any chafing or trauma. Skin is more susceptible to injury after
sunburn since UV radiation damages the skin cells, leading to cell death and disruption of the
structure, making it harder for the epidermis to heal from trauma. Since the dermis layer is also
impacted, loss of elasticity can occur since the collagen fibers are damaged, making the skin
more prone to bruising or tearing. Lastly, since this skin is healing, it is highly vascularized,
increasing the risk of bruising (Crane; Guerra, 2023). While Matt is healing from his burn, I
would advise him to stay out of the sun and moisturize his skin with aloevera or soy to soothe
the skin. I would also recommend that he stay hydrated, as the body cannot retain water as well
and can become dehydrated more quickly. Furthermore, he should not peel his skin or pop any
blisters as this is the body's natural response to prevent the skin from infection and rid the body
of dead skin cells (American Academy of Dermatology Association, 2024).
When Maria sees Matt after his vacation, she says that his sunburn is preparing his skin for a
deep tan and that he should go to this local tanning salon before any of the effects of the sunburn
wear off. Contrary to what many may think (including Maria), the tanning of skin is a sign of
skin damage, and the “golden hue” you get from a suntan is, in fact, the body's response to skin
injury from UV radiation (Santos-Longhurst, 2020) Therefore, the deep tan that Matt is
experiencing means his skin is severely stressed as the body produces more melanin to create
a darker appearance and reduce UV absorption to prevent any further damage. Any more
exposureto the sun or UV radiation would increase the damage to his skin, reverse any healing
that may have occurred, cause permanent skin damage, and lead to skin cancer (Santos-
Longhurst, 2020).
