human development class
ple” (p. 50). Rushton presented a vast array of scientific evidence in his concep- tualization, for example, a correlation of .62 between cranial capacity and distance from the equator with 20,000 crania rep- resenting 122 ethnically distinguishable populations (Beals, Smith, & Dodd, 1984). Templer and Arikawa (2003) re- ported a correlation of �.71 between mean IQ and mean high winter tempera- ture and a correlation of �.61 between mean IQ and mean low winter tempera- ture with 129 countries. There are alter- native explanations to those of Rushton for such findings. To relegate Rushton’s theory to the realm of absurdity, how- ever, would neither constitute optimal scientific reasoning nor represent an ideal spirit of scholarly disagreement.
REFERENCES
Beals, K. L., Smith, C. L., & Dodd, S. M. (1984). Brain size, brain morphology, climate, and time machines. Current Anthropology, 25, 301–328.
Rushton, J. P. (1995). Race, evolution and be- havior: A life history perspective. New Bruns- wick, NJ: Transaction.
Snyderman, M., & Rothman, S. (1988). The IQ controversy: The media and public policy. New Brunswick, NJ: Transaction Books.
Sternberg, R. J., Grigorenko, E. L., & Kidd, K. K. (2005). Intelligence, race, and genetics. American Psychologist, 60, 46 –59.
Templer, D. I., & Arikawa, H. (2003, Decem- ber). Temperature, skin color, per capita in- come, and IQ: An international perspective. Paper presented at the meeting of the Interna- tional Society for Intelligence Research, New- port Beach, CA.
Correspondence concerning this comment should be addressed to Donald I. Templer, Alliant International University, 5130 East Clinton Way, Fresno, CA 93727. E-mail: [email protected]
DOI: 10.1037/0003-066X.61.2.177
Examining Unproven Assumptions of Galton’s
Nature–Nurture Paradigm
Charles L. McLafferty Jr. Birmingham, Alabama
Sir Francis Galton’s (1869/1892) notion of nature versus nurture is a cornerstone of psychology: It was recently featured in two issues of the Monitor (March and April 2004) and was infused throughout the Jan- uary 2005 issue of the American Psychol- ogist. Sternberg, Grigorenko, and Kidd (January 2005) offered keen insights into
the pitfalls in the study of intelligence and race, discerning between folklore and sci- ence. Similar scrutiny is needed of the premise underlying these articles: that the nature–nurture paradigm is a scientific fact. Ultimately, the validity of statistical formu- lae derived from Galton’s thesis depends on unproven assumptions. Further, a di- mensional ontology allows expansion of the theoretical perspective.
The idea that nature and nurture make us who we are is easily distilled into a statistical formula: In terms of variance, “heritability and environmentality add to unity” (Sternberg et al., 2005, p. 53). The first assumption, termed exclusivity, stems directly from that idea: Only nature and nurture make us who we are. This leads to a paradigmatic requirement: No influences exist other than genetics and environment. The second assumption implicit in the body of nature–nurture research, termed univer- sality, is that the paradigm is valid for every human trait studied. An example will illuminate both assumptions.
A century ago, phenylketonuria (PKU) was purely heritable in terms of variability: The inability to metabolize phe- nylalanine resulted from a genetic defi- ciency. Fifty years ago, a scientist discov- ered its mechanism and prescribed a palliative diet deficient in phenylalanine. Today, PKU is cited as exemplary of na- ture–nurture interaction (e.g., Sternberg et al., 2005). However, a crack has appeared: Science has given each person the freedom to be tested and to follow the diet. Even for a physiological disorder such as PKU, na- ture and nurture do not account for all influences; the ability to choose likely ac- counts for most of the variability today, thus violating the exclusivity assumption. If one extends this example, on that theo- retical day when human DNA is com- pletely understood, Galton’s (1869/1892) paradigm will be refuted definitively: Sci- entists will have maximized human choice while minimizing adverse aspects of ge- netic inheritance.
If a physiological disorder might re- sult from more than nature and nurture, then what about psychological traits, in which the effects of human abilities such as choice, free will, responsibility, meaning, purpose, and spirituality may be magni- fied? If PKU provides even a single excep- tion to the universality assumption, then Galton’s paradigm must be validated for each trait studied.
A third assumption, complementar- ity, must also be addressed: Nature and nurture constitute a linear dichotomy, even in interaction. Note the wording of Sternberg et al.’s (2005) translation of
Galton’s paradigm quoted earlier: Nature and nurture “add to” one. As Sternberg et al. (2005) noted, “Heritability has a com- plementary concept, that of environmen- tality” (p. 53). This dichotomous struc- ture requires that variation from any other source automatically be included under heritability, environmentality, and/or their interaction, thus precluding its consid- eration outside of the paradigm (Biddell & Fischer, 1997). In the example of PKU, if heritability is minimized, is environmental- ity correspondingly increased? However, as mentioned earlier, the ability to choose likely has become the greatest source of variability.
A dimensional ontology allows a more parsimonious inclusion of these factors. Viktor Frankl (1967) noted that the person lives in three interpenetrating dimensions: soma (the physical body), psyche (the emotions and intellect), and the noëtic (the soul). The noëtic dimen- sion includes free will, responsibility, choice, spirituality, and the unique mean- ing capacity of Homo sapiens. Although animals share soma and psyche with hu- mans, the noëtic is defined as that which differentiates us from animals (Frankl, 1967). In this ontology, genetic expres- sion is somatic in origin— genes synthe- size amino acids, pure and simple; nur- ture occurs in both soma and psyche. From this perspective, nature and nurture are dimensionally different rather than complementary.
The existence of dimensional causes other than heritability and environmentality violates all three assumptions. Theorists have proposed factors that operate in con- junction with genetics and environment, such as symbol systems (language, science, math, musical notation; Gardner, Hatch, & Torff, 1997) and human agency (choice, free will; Biddell & Fischer, 1997; Frankl, 1967). Assuming that animals do not have language, science, and math, nor the choice and free will needed for agency, these dis- tinctly human variables are noëtic (Frankl, 1967). How do they affect nature–nurture research?
Studies of twins with regard to lan- guage development and IQ invariably in- volve the measure of noëtic symbol sys- tems, using the manipulation of language and/or representations of math or logic. Similar studies of substance abuse ignore the noëtic dimension: The most successful treatment involves 12-step programs that invoke a “Higher Power” to strengthen the ability to choose. Twin studies of career selection paradoxically assume that only nature and nurture determine choice, pre- cluding the Jungian sense of “finding one’s
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calling.” For each of these fields of study, the noëtic dimension is a confounding vari- able, its variability subsumed into genetic and/or environmental components.
From a clinician’s standpoint, the small arena of choice is likely the most powerful fulcrum available for change. The healer’s role is arguably to transform the client’s sense of agency from “insignifi- cant” into a force capable of harnessing and transcending nature and nurture. With choice come responsibility and the realiza- tion of freedom, which cannot occur with- out meaningful and purposeful options. In- deed, it is through exercising our ability to choose that we become fully human. Gal- ton’s (1869/1892) paradigm ultimately as- sumes that the person has no true freedom. To rephrase Frankl (1967), nature and nur- ture represent our fate, whereas the noëtic represents our capacity to take a stand to- ward that fate.
For decades, Galton’s (1869/1892) nature–nurture paradigm has anchored psy- chological research and theory. Though re- nowned, his idea and its derivations were never scientifically validated. Galton’s idea, and all studies based on it, bear re- consideration. Inclusion of the noëtic di- mension and its intercourse with genetics and environment allows the researcher to consider the person in studies of human development.
It is time for a new paradigm: Nature, Nurture, and the Noëtic.
REFERENCES
Biddell, T. R., & Fischer, K. W. (1997). Be- tween nature and nurture: The role of human agency in the epigenesis of intelligence. In R. J. Sternberg & E. Grigorenko (Eds.), Intel- ligence, heredity and environment (pp. 193– 242). Cambridge, England: Cambridge Uni- versity Press.
Frankl, V. E. (1967). Psychotherapy and exis- tentialism. New York: Washington Square Press.
Galton, F. (1892). Hereditary genius: An inquiry into its laws and consequences. Retrieved No- vember 21, 2005, from http://www.mugu.com/ galton/books/hereditary-genius/index.html (Original work published 1869)
Gardner, H., Hatch, T., & Torff, B. (1997). A third perspective: The symbol systems ap- proach. In R. J. Sternberg & E. Grigorenko (Eds.), Intelligence, heredity and environment (pp. 243–268). Cambridge, England: Cam- bridge University Press.
Sternberg, R. J., Grigorenko, E. L., & Kidd, K. K. (2005). Intelligence, race, and genetics. American Psychologist, 60, 46 –59.
Correspondence concerning this comment should be addressed to Charles L. McLafferty Jr., 3603 Lorna Ridge Drive, Birmingham, AL 35216. E-mail: [email protected]
DOI: 10.1037/0003-066X.61.2.178
Racing Toward the Finish Line
Robert J. Sternberg Tufts University
Elena L. Grigorenko and Kenneth K. Kidd Yale University
Carey (2006, this issue) cited in his re- sponse to our article (Sternberg, Grigor- enko, & Kidd, January 2005) a study by Tang et al. (2005) showing that “of 3,636 subjects of varying race/ethnicity, only 5 (0.14%) showed genetic cluster member- ship different from their self-identified race/ethnicity” (p. 268). Carey (2006, p. 176) then stated that he
would very much like to hear a response to this finding from Sternberg et al. (2005), who main- tained that “race is a socially constructed con- cept, not a biological one” (p. 49), that reifies those physical correlates of ancient population dispersions “as deriving from some imagined natural grouping of people that does not in fact exist, except in our heads” (p. 51).
We are glad to provide a response. Imagine the case of Dr. Smith. Dr.
Smith has a hypothesis that the proper basis on which to classify people’s race is not skin color, which has so often been used (mistakenly, he believes), but rather eye color. He discovers some interesting facts. Whereas 0.14% of a sample showed ge- netic cluster membership different from their self-identified race/ethnicity based on skin color and other such criteria, not even one person misidentified genetic member- ship on the basis of eye color. He is im- pressed. He then discovers that race de- fined by eye color is quite powerful. Blue- eyed races tend to live more in Northern climes, and brown-eyed races, closer to the equator. Blue-eyed people, on average, are more susceptible to sunburn and concomi- tants of it. Blue-eyed races have health problems, on average, more typical of peo- ple in Northern climes, and brown-eyed races, those more typical of equatorial climes. Although the effects are not whop- ping, they are statistically significant. There are even differences, on average, in the distribution of languages these races speak. Dr. Smith even notes that, in earlier times, eye color actually was used as a basis for classifying people on the basis of race, which gives historical weight to his scientific finding. (As an example, Stern- berg’s mother escaped Nazi-occupied Aus- tria only by virtue of her blue eye coloring, which suggested “Aryan” racial member- ship to police guards on a train.) The point
of the apocryphal story above is that cor- respondence between self-identification and biological characteristics does not form a suitable basis for assigning people to “races.”
Moreover, it appears that Carey’s (2006) response to our article can be ex- plained, at least partially, by his incomplete understanding of the data, analyses, and results presented in Tang et al. (2005). First, the Tang et al. study was done using samples primarily from the United States, where the primary ethnic groups are de- rived from very different parts of the globe: European (primarily Western European), African American (of mixed West African and European origin), East Asian (Chinese, Japanese, Vietnamese, and other groups not generally distinguished by U.S. policy- makers but genetically distinguishable), and Hispanic Mexican American (of mixed Native American and European origin). Thus, there exist noticeable genetic differ- ences that parallel the origins and culture differences among these groups, specifi- cally, the immigration patterns from differ- ent parts of the globe into the United States. Correspondingly, given where these cultur- ally defined groups came from geographi- cally, it is not surprising that they have genetic differences. More relevant to the point that we make—that is, of the conti- nuity of genetic diversity among various ethnic groups and, correspondingly, the ar- tificial nature of the concept of race—is the observation that African Americans can have considerable differences in their an- cestral contributions, depending on where, culturally and geographically, their ances- tors came from. In the United States, cul- turally (socially) and historically, anyone with any African ancestry is viewed as or referred to as “African American.” Con- sider the children of Thomas Jefferson and Sally Hemings. She was already half Eu- ropean (a half sister of Thomas Jefferson’s wife), and their children were therefore three quarters European, yet they were still socially slaves and “African American.” Depending on how such individuals self- identify and how the genetic clusters are derived statistically, they will cluster with “pure” Europeans or a “partially African” cluster and will either fit or be outliers for these clusters.
Second, we also note that the analysis in Tang et al. used the “no admixture” option; in other words, the analyses did not allow for a careful examination of sources and structure of variability “added” by mixed ethnical marriages, which, clearly, would minimize estimates of within-group variation and maximize estimates of be- tween-groups variation. Third, despite the
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