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Please post 2 or more peer responses

In the response posts, remember to demonstrate you have read and understood the student's post by taking their discussion to the next level. Include the following in your responses:

· State whether you agree with the meaning of the relationship as described by your peer.

· Can you think of other variables that might help explain the relationship between the two variables they chose? 

· If no relationship was found, can you offer any reasons why not?

· Were the relationships they discovered in their research strong enough to use to predict one variable from the other?

· What is the real-world significance of the findings they described?

Please be sure to validate your opinions and ideas with citations and references in APA format.

Estimated time to complete: 2 hours

Response posts are worth 50% of your grade for this discussion. Please review the post and response expectations Please review the rubric to ensure that your response meets the criteria.

I am a nursing student so I chose the variable; weight and blood pressure. I will explain the correlation between the two and the continuous affect on the body. When a person gains weight or has a high BMI for their age and gender, their body must increase its function to support homeostasis. This simply means that when a person gains or increases their BMI, they have more muscle and muscle will require more oxygen demands since it is a vital part of our body. The more oxygen your body requires, the more perfusion increases. Perfusion is the circulation of blood due to its ability to carry oxygen to out organs. Increasing blood flow throughout the body means an increase in workload for the heart. Cardiac output will increase to meet the demands of the body as it is stimulated by the sympathetic nervous system. This increase and stimulation rises systolic and diastolic blood pressure. Another example is the more weight a person carries, the more damage the arteries sustain due to the pressure of blood flowing to reach organs. Arterial damage means the blood is restricted to reaching the organs. A major issue with that is the kidneys because the kidneys help regulate blood pressure. Kidneys remove fluids and waste and produce hormones that control blood vessel constriction. 

Reference:

El Meouchy, P., Wahoud, M., Allam, S., Chedid, R., Karam, W., & Karam, S. (2022). Hypertension Related to Obesity: Pathogenesis, Characteristics and Factors for Control.  International journal of molecular sciences23(20), 12305. https://doi.org/10.3390/ijms232012305

Khalid, F., Siddique, A., Siddiqui, J. A., Panhwar, G., Singh, S., Anwar, A., & Hashmi, A. A. (2020). Correlation Between Body Mass Index and Blood Pressure Levels Among Hypertensive Patients: A Gender-Based Comparison.  Cureus12(10), e10974. https://doi.org/10.7759/cureus.10974

Kim H. L. (2023). Arterial stiffness and hypertension.  Clinical hypertension29(1), 31.  https://doi.org/10.1186/s40885-023-00258-1Links to an external site.

Suzumoto, Y., Zucaro, L., Iervolino, A., & Capasso, G. (2023). Kidney and blood pressure regulation-latest evidence for molecular mechanisms.  Clinical kidney journal16(6), 952–964. https://doi.org/10.1093/ckj/sfad015

The topic of my research is regarding maternal obesity and gestational diabetes and its effect on the child. As nurses, monitoring weight and blood glucose levels are standard practices especially in the world of obstetrics. Though these two variables are directly affecting the mother, they can tell us a great a deal of the mother’s and baby’s health and in this case the future health of the child. Obesity affects the body in various ways such as extra pressure on bones and joints, increased breathing difficult, increased inflammation and chemical changes in the body. Obesity tends to lead to insulin resistance which leads to ineffectively absorption of excess glucose. Thus, allowing the liver to convert the extra glucose to fat. Increased glucose levels also known as hyperglycemia can affect the body by causing nerve damage, making the body harder to heal, as well as possibly causing further chemical changes in the body. The risk of childhood obesity begins before birth. The most common prenatal causes of childhood obesity include maternal gestational diabetes mellitus (GDM) and pre-pregnancy obesity (PPO) (Choi et al., 2022). GDM also increases the risk of pre-eclampsia, caesarean section, and up to 50% of women will develop type 2 diabetes later in life. From a fetal point of view, it increases the risk of macrosomia, shoulder dystocia and birth trauma (Poblete, & Olmos, 2021). A statistically significant difference was found in the prevalence of childhood obesity depending on the presence of PPO or GDM in mothers. Children whose mothers had both PPO and GDM had a significantly higher obesity prevalence and BMI percentile across all age groups (Choi et al., 2022). Research by Kim and Ayabe (2023) supports that intrauterine exposure to maternal obesity can lead to adverse health outcomes in the offspring, including an increased incidence of metabolic syndrome and obesity in the child. Based on these results, we can conclude that there is a correlation between mothers who have PPO and GDM, as they are likely to have children who have higher BMIs compared to mothers who were not obese or did not suffer from gestational diabetes. The correlation between variables measures both the direction and the strength of tendency to vary together (Frost, 2024).

 

 

References

Choi, M. J., Yu, J., & Choi, J. (2022). Maternal pre-pregnancy obesity and gestational diabetes mellitus increase the risk of childhood

obesity. Children (Basel, Switzerland), 9(7), 928. https://doi.org/10.3390/children9070928

Frost, J. (2024, April 30). Interpreting correlation coefficients. Statistics By Jim. https://statisticsbyjim.com/basics/correlations/

Kim J, & Ayabe A. (2023). Obesity in Pregnancy. StatPearls. Treasure Island (FL): StatPearls Publishing; 2024 Jan-. Available from:

https://www.ncbi.nlm.nih.gov/books/NBK572113/

Poblete, J. A., & Olmos, P. (2021). Obesity and gestational diabetes in pregnant care and clinical practice. Current vascular pharmacology, 19(2),

154–164. https://doi.org/10.2174/1570161118666200628142353