neurobiology of disease

profilesabrinak2751
nerubiomidterm.pdf

1.Compare the "normal" function of the PFC with its proposed function in an individual with ADHD. 2 paragraph minimum. 

2.Explain the dopamine hypothesis of ADHD. 1 paragraph minimum. 

3.Explain the dopamine hypothesis of ADHD. 1 paragraph minimum. 

4.Why would a reduced size of the corpus callosum be problematic, with reference to autism? 1 paragraph minimum. 

5.5 a. What are mirror neurons? (1 point - 1 paragraph minimum) 5 b. How may mirror neurons play a role in autism? (1 point - 1 paragraph minimum)

6.Explain the underlying pathogenesis of Duchenne's Muscular Dystrophy (DMD). 1 paragraph minimum. 

7.Based on the information you've read about DMD, what are some daily life limitations, postures, and weaknesses you'd expect to see in the clinical presentation of a patient with DMD? 2 paragraph minimum

8.Compare/contrast a complete vs. incomplete spinal cord injury.

9.Compare/contrast an individual and their impairments with an C1-C4 injury vs. an injury in the lumbar spine.

10.Explain how the vascular system of the spinal cord is injured in SCI secondary injury. 1 paragraph minimum.

11.Discuss the pros and cons of the glial scar in SCI. If you use a list, be sure to explain each bullet point.

12.Explain why it is difficult to diagnose the level of injury, immediately after SCI. 1 paragraph minimum.

13.Give an example of a coup-counter coup TBI: Discuss the mechanism of injury (how the injury happened), which location of the brain is considered the coup injury, which location is considered the counter-coup injury, and the resulting possible functional deficits based on injury locations.

14.Explain why the release of glutamate and Ca2+ so problematic after TBI. 2 paragraph minimum.

15.Explain why the release of glutamate and Ca2+ so problematic after TBI. 2 paragraph minimum.