Case Study TH

EEE1212
RadionuclideTherapies-Notes2.pptx

Radioimmunotherapy (RIT) and radionuclide therapy

RIT- Radioimmunotherapy

Objective of RIT is to deliver a lethal radiation dose to tumor cells

Effective due to Antigen-Antibody reaction

Maximize dose to tumor while minimizing dose to normal tissue

Primary limiting factor = bone marrow depression

Decrease in blood cells

Noticeable in 2-3 weeks

Lowest at 4-8 weeks

Recovery at 12 weeks

RIT

Informed consent is important for therapy and includes:

Pregnancy/lactation

Radiation safety post dose/written instructions

Risk of cytopenia

Risk of hypothyroidism (Bexxar™)

Development of secondary tumor/leukemia

HAMA response

RIT

Human Anti-Mouse Antibody (HAMA) Response

Antibody treatment is a type of therapy that is used to treat certain types of cancer and immune disorders

Antibodies are proteins naturally formed by the body in response to a foreign substance - known as an antigen

Antibodies can also be grown outside the patient’s body and then injected to help aid the immune system to fight disease - called monoclonal antibodies because they are created to target one specific antigen

HAMA

For decades mice were used extensively in the production of monoclonal antibodies

But the treatments were not as effective as doctors had hoped

Patients reacted to the mouse antibodies as if they were a foreign substance, and created a new set of antibodies to the mouse antibodies

Doctors have termed this the “HAMA response,” referring to the development of Human Anti-Mouse Antibodies (HAMA)

HAMA

The HAMA response is essentially an allergic reaction to the mouse antibodies that can range from mild (rash) to extreme and life-threatening (renal failure)

HAMA can also decrease the effectiveness of the treatment or create a future reaction if the patient is given a subsequent treatment containing mouse antibodies

Approximately one-third to half of patients receiving mouse-derived antibodies will develop some form of HAMA response

At least ten percent of the general population has been observed to carry some form of animal-derived antibodies, most often from mice due to the preponderance of medical agents made from the serum of animals

Zevalin™

1st Murine Antibody approved to treat CA

Extends life by inducing remission

Improves quality of life by reducing symptoms of disease

Zevalin™

Ibritumomab Tiuxetan

Ibritumomab = murine antibody parent of rituximab (Rituxan®)

Protein which interferes with growth and reproduction of lymphomas and leukemia cells

Tiuxetan = chelator that binds to RN

111In (imaging)

HL = 67.2 hr

Gamma = 245 & 172 keV

90Y (therapy)

HL = 64 hrs

β- = 2.28 MeV (Shackett)

940 kev + neutrino

Zevalin™

Initially approved for relapsed or refractory:

Low grade/Follicular or transformed B-cell Non-Hodgkins Lymphoma (NHL)

2002- FDA approved for 1st RIT drug for cancer

2009- FDA approved for untreated NHL

≈95% B-cell lymphomas have CD20 antigen on surface of cell

Zevalin™ targets CD20 antigen

Zevalin™

Contraindications

Pregnancy/nursing

Platelets < 100,000

Neutrophils < 1500

≥ 25% lymphoma marrow involvement (impaired bone marrow reserve)

Bone marrow transplant

Known hypersensitivity to murine proteins

Altered biodistribution as determined by 111In-Zevalin images

Zevalin™

No patient prep

Outpatient

Check for blood thinners:

Increased risk of bleeding due to an increased risk of thrombocytopenia (low platelet count)

Warfarin (coumadin)

Aspirin

NSAIDs (Non-steroidal anti-inflammatory drugs)

Ibuprofen, naproxen

Motrin, Advil, Aleve

Zevalin™

Day 1

250mg/m2 of Rituxan® via slow IV infusion

Begin infusion rate at 50mg/hr

If no reaction, increase rate in 50mg increments every 30 min up to 400mg/hr

Bolus may result in death w/in 24 hrs (MI, hypoxia, anaphylaxis)

If reaction occurs, decrease infusion rate by 50%

If severe reaction; stop- treat- restart at 50%

Onset may begin ≈ 30-120min

Zevalin™

Rituxan® saturates normal CD20 sites (spleen, circulating B cells)

Increases amount of RA Zevalin™ targeting tumor cells

Without Rituxan® ≈ 18% RP uptake

With Rituxan® ≈ 56-92% RP uptake (varies upon infusion rate)

Zevalin™

Day 1(Not currently required)

Within 4 hrs of Rituxan®, infuse 5mCi 111In Zevalin™ over 10 min IV push

Flush IV to ensure full dose

Whole body images taken at 2-24 hours and again at 48-72 hours

May need 90-120 hr imaging

Depending on biodistribution

Zevalin™

Organ Scan 2-24 hours Scan 48-72 hours Scan 90-120 hours (optional)
Blood pool Present Decreases Decreases
Liver and Spleen Moderate high-high Moderate high-high Moderate high-high
Kindeys, Bladder, Bowel Moderate low-very low Moderate low-very low Moderate low-very low
Tumor Variable Variable Variable

Expected Bio-distribution

Zevalin™

Organ Scan 2-24 hours Scan 48-72 hours Scan 90-120 hours (optional)
Blood Pool Not Visualized Not Visualized Not Visualized
Lungs > Cardiac Blood Pool Difuse uptake > Liver Diffuse uptake > Liver
Kidneys > Liver in posterier view > Liver in posterier view > Liver in posterior view
Bowel > Liver > Liver > Liver

Altered Bio-distribution

Zevalin™

Imaging evaluates biodistribution of RP

Tumors may be less evident in abnormal biodistribution

Concerns about radiation to non-target organs if altered

Normal distribution via In-111 Zevalin imaging was previously required before starting therapy

Was only a requirement in US, Switzerland, and Japan

42 other countries approved the therapy without In-111

NO LONGER REQUIRED IN THE UNITED STATES- Nov 2011

Zevalin™

Expected Biodistribution Observed

Blood pool visible on early images less on delayed

Moderately high-high liver on early and delayed

Moderately low-low in kidney, bladder and bowel in early and delayed

Tumor uptake in left axilla, mesenteric lymph nodes, bilateral pelvis, testicular regions

Zevalin™

Day 7-9

Therapy regimen

2nd Rituxan® infusion (same rate)

Within 4 hrs, 90Y Zevalin™ infused over 10 min

Dosage = 0.4 mCi/kg if platelets ≥ 150,000

Dosage = 0.3 mCi/kg if platelets 100,000-149,000

Zevalin™

Typical dosage = 21-30 mCi

Can not exceed 32 mCi

Assures:

No normal organ receives > 2000 rads

No red bone marrow receives > 300 rads

Zevalin™

Approximately 73-83% patients responded to therapy

Approximately15-51% remained in remission lasting ≈ 23 months

Side effects:

GI symptoms, cough, dizziness, anxiety

Delayed myelosuppression

Zevalin™

Blood/platelet counts monitored ≈ 12 weeks

≈ 29% develop infections (low WBC)

≈ 7% need hospitalization (infections)

Bleeding/bruising (low platelets)

Fatigue (low RBC)

HAMA ≈ 2%

Zevalin™

No isolation from family required

No patient specific calculations needed (pure β-)

Dose to others < 1 mrem (due to brems)

Return to work immediately in most cases

Avoid body fluid contact for about 1 week following therapy

Bexxar™- Discontinued Feb 2014

RIT

Tositumomab

131I

NHL

Procedure includes both Dosimetric and Therapeutic steps

Bexxar™

Patient requirements:

> 50,000/mm3 platelet counts

Absolute Neutrophil Count > 800/mm3

Bexxar™

Patient Prep:

Pre-dose with 650 mg Acetaminophen and 50 mg Antihistamine

30 min prior to dosing with non-radioactive tositumomab

Reduces side effects

Bexxar™

Daily Iodine supplement to block thyroid uptake of 131I

SSKI (4 drops orally 3x per day)

Lugol’s (20 drops orally 3x per day)

KI (130 mg daily)

≈ 9% become hypo

Start 24 hours pre-dose

Continue 2 weeks post dose

Bexxar™

Dosimetric step

450 mg “cold” tositumomab in 50 ml normal saline

Distributes RP

IV infusion over 60 min

5 mCi 131I Bexxar™ in 30 ml normal saline

IV infusion over 20 min

Bexxar™

WB image post infusion

Determines localization in body

Day 2-6:

WB image (check for residual activity)

Used to calculate therapy dosage

Bexxar™

Day 7-10:

Infuse cold tositumomab over 60 min

Infuse 131I Bexxar™ therapy dosage over 20 min

Dosages = 30-200 mCi

Bexxar™

Side effects:

Flu-like symptoms

1-2 days post dose

Bruising/bleeding

3-4 weeks (decreased platelet ct.)

Allergic reaction (rash, swelling, cough)

Antihistamine helps in limiting symptoms)

≈ 10% HAMA

Bexxar™

Hospitalization due to radiation exposure

Sent home if dose specific calculation done

Give instructions on Rad Safety – similar to thyroid ablation restrictions

Public safety issue due to high energy gamma

Normal Hct

Polycythemia Radionuclide Treatment

Many types of polycythemia disorders

Polycythemia Vera (primary, true)

Impacts 5-26 people/1million/year

Chronic neoplastic disease of bone marrow

High RBC ( Hematocrit [Hct] > normal)

May have high WBC ( >12,000/ μL)

May have high platelets (>400,000/μL)

Average age of onset ≈ 60

Survival rate:

1.5 years without therapy

10 years with therapy

Not effective on other forms of polycythemia

Secondary Polycythemia

Over stimulation of bone marrow

Increase of RBC production due to increase of erythropoietin

Relative Polycythemia

Normal RBC, but decreased plasma volume

Can be treated by bleeding

Polycythemia Radionuclide Treatment

Treatment:

Phlebotomy (nearly always performed)

Chemotherapy (preferred treatment along with phlebotomy)

Radiation therapy (secondary treatment)

Untreated:

Slow circulation (thick blood)

Local hypoxia

Decreased blood flow to vital organs

Thrombosis

Polycythemia Radionuclide Treatment

Polycythemia

32P sodium phosphate

Same as used for bone mets

Was first used to treat leukemia

Not as common

Dose = 2.3 mCi/m2

3-5 mCi with 10 ml normal saline

IV butterfly

30-60 sec injection

Polycythemia

≈ 85% RP selectively concentrates in mitotically active cells of bone/bone marrow

Incorporated into DNA in cells

Direct damage due to β-

Polycythemia

Results:

Blood chemistry normal within 4-6 weeks

Remission ≥ 2 years

10-20% develop acute leukemia

No remission:

Increase dose by 25%

Check blood levels at 3 months

Increase dose by 25% again, check at 3 months

Do not exceed 7 mCi within 6 months!!!

Malignant Effusions Therapy

Effusion:

Abnormal collection of fluid in a cavity

Due to:

Increased fluid production (serosal irritation)

Decreased fluid reabsorption (lymphatic obstruction)

Malignant Effusions

Pleural

Primaries: Breast, lung, ovarian, lymphomas

Nonmalignant: infections, cirrhosis, cardiac, TB

Peritoneal – Ascites

Primaries: ovarian, renal, GI

Nonmalignant: cirrhosis, peritonitis, CHF

Pericardial

Primaries: Lung, breast, lymphomas, melanomas

Nonmalignant: infection, radiation induced pericarditis

Malignant Effusion

Therapy

Palliative treatment of symptoms

Prevention of recurring disease

Other therapies

Centesis (puncture fluid removal)

Chemotherapy

Malignant Effusion

32P chromic phosphate (colloid)

Dose depends on cavity

Peritoneal = 10-25 mCi in 30-500 ml normal saline

Pleural = 6-15 mCi in 30-50 ml normal saline

Pericardial = 5-10 mCi in 30-50 ml normal saline

Malignant Effusion

Insert catheter and remove fluid

Administer RP into organ

1-3 mCi 99mTc SC may be added for imaging

Remove catheter, add dressing for leakage

Change positions every 10-15 min for 2 hrs

Distributes RP

Malignant Effusion

RP taken up by macrophages in cavity

RP adsorbs to serosal lining

β- energy deposited in tissue

Fibrosis of mesothelium and small blood vessels

Decrease fluid production results

Malignant Effusions

Cavity leakage ≤ 10%

Regional lymph node uptake ≈ 2%

< 5% remains free @ 24 hrs

Image using Brems (160-450 keV) or added 99mTc SC

Malignant Effusions

Results take about 3 months

Decrease pain

May repeat dosing

Contraindications:

Loculated fluid

Pocket of fluid walled off from other fluid

Expected live span < 6 months

32P Radiation Safety

Patient may be hospitalized due to medical reasons, not radiation hazard

External exposure levels low

Why??

Minimal exposure due to Brems

Leakage, contaminated gauze

Synovitis Radionuclide Therapy

Inflammation of synovial membrane

Causes:

Rheumatoid Arthritis – autoimmune disease

Bleeding into joint: hemophilia

Nodules projecting into cavity

Synovitis

Results:

Increased secretion of synovial fluid

Release of enzymes/cell factors that cause destruction of cartilage/joints

Local irradiation:

Decrease fluid production/pain

Destroys cells that release enzymes that cause joint destruction

Synovitis

RP used depends on joint size and synovial thickness

Large, stable particles (phagocytized by synovial macrophages)

32P chromic phosphate- Phosphocol (Colloid)

Knees and elbows

90Y

Large joints, knees with thick synovia

186Re (Rhenium)

Intermediate joint sizes

169Er (Erbium)

Small joints

Synovitis

Typical dose range for 32P:

Knees = 0.5-1mCi

Elbows/wrists = 150-500 µCi

Depends on size of joint involved

Synovitis

Possible Patient Prep:

Factor VIII – hemophiliacs given anti-hemophilic factor (AHF) pre-therapy, as well as 24 and 72 hrs post therapy

Betadine – prep for spinal tap

1% lidocaine – topical anesthetic

Synovitis

Place needle in synovial space (fluoro, U/S)

Connect tubing/stopcock

Attach syringe/aspirate fluid

Inject 1mCi 99mTc SC for image (or use Brems)

Inject RP

Attach syringe with 1% lidocaine

Synovitis

Inject lidocaine along needle tract while needle is withdrawn

Helps prevent inflammation/leakage of dose

Flex joint to distribute RP

Immobilize joint for 48 hrs (minimizes leakage)

GM meter to assess nodal uptake (≈ 2%) at 24 hrs, 72 hrs, and 7 days

Synovitis

Results:

Decreased pain/inflammation in joint

Decreased degradation of cartilage/bone in joint

Increased joint function

78-84% improvement in 3-6 months

Radiation dermatitis @ injection site

Prostate CA Bone METS Treatment

Xofigo ®

Used for treatment of prostate CA that is resistant to medical or surgical treatments that lower testosterone, or bone metastases of prostate CA

Termed Castration-Resistant Prostate Cancer (CRPC) or Metastatic Castration-Resistant Prostate Cancer (mCRPC)

223Radium

11.43 day half-life

4.78 MeV alpha emitter

Debate over therapeutic or palliative value

Xofigo ®

Dosage

1.35 mCi per kg

One dose every 4 weeks for 6 cycles

Slow IV injection for 1 minute

Flush IV line with 10 ml saline before and after

Pharmacology

Mimics calcium and forms complexes with bone mineral hydroxyapatite at areas of increased bone turnover

Excretion:

48 hours post dose, fecal excretion at 13%

7 days post dose, 63% excreted via feces

Xofigo ®

Precautions:

Advise male patients who are sexually active to use condoms and female partners to use highly effective contraceptive method for 6 months after treatment

Myelosuppression may occur

Hydration is important

Bathroom hygiene - Flush toilet several times, wash hands thoroughly

Xofigo ®

Contraindications

Pregnant?

Other important details:

No outpatient restrictions (alpha emitter)

Common side effects include:

Nausea

Diarrhea

Vomiting

Peripheral edema

Low blood counts

Just the beginning…

Research still continuing in radionuclide therapies

Possible avenues of therapy:

More Alpha-emitting particles

Peptide Receptor Radionuclide Therapy (PRRT)

Hormone-Delivered Radiotherapy

Alzheimer’s treatments

QUESTIONS?